More evidence of vaccine negative effectiveness and mounting evidence of infection complications seen in vaccinated individuals. Most importantly, we summarise the author’s 5 ways how to help protect the immune system of patients to minimize risks.

Described content:
Infections post-surgery:
https://virologyj.biomedcentral.com/articles/10.1186/s12985-022-01831-0

Lance negative VE: https://www.thelancet.com/journals/lancet/article/PIIS0140-6736(22)00089-7/fulltext

Immune system protection: https://www.termedia.pl/Five-important-preventive-measures-against-exacerbation-of-coronavirus-disease,118,44963,1,1.html

Lipid emulsion risks: https://www.termedia.pl/Risk-of-propofol-use-for-sedation-in-COVID-19-patient,118,42287,1,1.html

“COVID-19 – Q and A for the People#14” event on ZOOM:
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Jennifer Heimall, MD, from the Division of Allergy and Immunology at CHOP answers the question can people with immunodeficiencies get the COVID-19 vaccine.
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Atopic dermatitis (Eczema): Symptoms, causes, triggers and treatments (dermatology lectures). This video is available for instant download licensing here : https://www.alilamedicalmedia.com/-/galleries/all-animations/skin-and-hair-videos/-/medias/64bb948d-45ab-4e66-bf7a-90e417e8135e-eczema-narrated-animation
Voice by: Sue Stern
©Alila Medical Media. All rights reserved.
Support us on Patreon and get FREE downloads and other great rewards: patreon.com/AlilaMedicalMedia
All images/videos by Alila Medical Media are for information purposes ONLY and are NOT intended to replace professional medical advice, diagnosis or treatment. Always seek the advice of a qualified healthcare provider with any questions you may have regarding a medical condition.
Eczema, or dermatitis, is a group of conditions characterized by inflammation of the skin. Among the many types of dermatitis, the most common is atopic dermatitis, also known as atopic eczema. Very often, when not specified otherwise, the term “eczema” is used to describe the atopic type.
Symptoms of atopic dermatitis include rashes, redness, scaling, and occasionally small blisters. Depending on the patient’s age, these patches may appear on the face, scalp, neck, inside the elbows, behind the knees, on the buttocks, hands and feet. The condition evolves in the form of recurrent inflammatory flare-ups followed by periods of remission. Flare ups can be triggered upon contact with irritants such as soap, detergents, rough fabric or certain foods. A dry atmosphere, changes in temperature, dental eruptions and stress are also common triggers. Over time, the skin can become thickened, bumpy and constantly itch, even when the inflammation is not flaring up. Atopic eczema usually starts in early childhood and MAY last into adult life. Most children outgrow the disease with age but their skin may remain dry and easily irritable.
Atopic dermatitis is an allergic disease. The cause is unknown but it is likely to involve genetic and environmental factors. Atopic eczema often runs in families whose members also tend to develop hay fever, asthma and certain food allergies. Most notable is the gene that encodes for filaggrin, a protein involved in water retention and is responsible for the skin barrier function. Mutations in the filaggrin gene cause dry skin and, as a result, a strong susceptibility to the disease. Eczema is NOT contagious.
There is no cure for atopic dermatitis. Treatments aim to relieve symptoms, reduce frequency of flare ups and prevent skin infection. A treatment plan may include:
– Lifestyle changes: bathe at least once a day but avoid soaps; wear silk clothing and avoid wool; avoid allergy triggers.
– Skincare: use oil-based, fragrance-free moisturizers to keep the skin hydrated during remissions.
– Medications: anti-inflammatory drugs such as steroid creams can be used during flare-ups. Antibiotics may be required if skin infection occurs.
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What is rheumatoid arthritis? Rheumatoid arthritis is an autoimmune condition where the immune system attacks the joints.

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My goal is to reduce educational disparities by making education FREE.
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Discover if eczema is associated with a weak immune system. This video looks at why sleep deprivation may worsen skin symptoms for more information and tips that may help soothe the skin and improve sleep read my article here: https://www.avogel.co.uk/health/skin/eczema/does-eczema-mean-you-have-a-weak-immune-system
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Normally, immune cells detect and attack cancer cells. But some cancer cells can avoid detection or block an attack. Cancer treatments called immunotherapies help immune cells better detect and kill cancer.

This video has open captions.
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After a lifetime of debilitating infections, Tara Sakraida Parker found answers—and treatment—at Johns Hopkins, through immunologist Antoine Azar and the Center for Adult Primary Immunodeficiency Center.

Visit us: https://www.hopkinsmedicine.org/news/publications/hopkins_medicine_magazine/issues/winter-2019
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Immunodeficiency means immune system is unable to protect the host from disease causing agents or from malignant cells.

#agaricus #immunodeficiency #immunodeficiencydisorders #primaryimmunodeficiency #secondaryimmunodeficiencies

PRIMARY IMMUNODEFICIENCY DISORDERS
https://youtu.be/-azY18Nz6u4

DENDRITIC CELL
https://youtu.be/DJ5On8GRwZw

AGRANULOCYTES
https://youtu.be/fMTpLypZsXA

GRANLOCYTES
https://youtu.be/YFeUI2-jiAY

PHAGOCYTOSIS
https://youtu.be/IoV7f_Iz5-U

Reference
https://amzn.to/3L6VASR
https://amzn.to/37Qg8AJ

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This is an overview of primary immunodeficiency disorders.

Graphics were created or adapted from Wikimedia Commons.
This presentation was created using Google Slides.

ADDITIONAL TAGS:
Selective IgA deficiency Hyper IgM syndrome X-linked Bruton’s agammaglobulinemia Common variable immunodeficiency C1 esterase inhibitor 22q112 deletion / DiGeorge Severe combined Wiskott–Aldrich Ataxia–telangiectasia Chronic granulomatous disease Leukocyte adhesion Chédiak–Higashi IgE Job Terminal complement C5-C9 Primary disorders B-cell humoral immunity deficiencies T-cell cellular lymphocyte and cellular phagocytosis Most common primary antibody Dx ↓ levels everything else normal normal IgG IgM B cells Related to CVID → similar arrest of cell differentiation Recurrent mouth airway respiratory infxns GI Giardia treat with abx Recall – mucus membranes still works so most patients are healthy asymptomatic undiagnosed benign Rarely presents severe rapid seconds minutes anaphylactic reaction following transfusion blood or IVIG Pathophys Anti-IgA antibodies against the new foreign Stop transfusion Give IM epi +/- bronchodilators antihistamines pressors mechanical ventilation If necessary give IgA-depleted Antibody class switching differentiation doesn’t work Cause absence CD40 ligand ↑ IgA nl cells Minimal nonselective defense Increased susceptibility recurrent sinopulmonary infxns Viruses encapsulated bacteria Treatment scheduled XLA recessive males only BTK gene mutation defective Bruton tyrosine kinase impaired maturation Ig production Signs/symptoms sinopulm Deficiency in opsonizing IgG otitis sinusitis pneumonia encapsulated Pseudomonas S pneumo Haemophilus Symptoms begin at 3-6 months transplacental maternal fades Small lymphoid tissue tonsils adenoids spleen lymph nodes enteroviral infxn Deficient response Diagnosis IgE Confirm flow cytometry B-cells low might say CD20+ low T-cells normal/high SCIG IVIG maybe prophylactic CVID abnormal plasma Decreased Presents adults children 15-35 yo as early puberty Males females resp lung sinus ear salmonella Campylobacter Giardia presentation lungs bronchiectasis fibrosis IBD-esque chronic diarrhea Less form XLA Need least two No vaccinations Upper lower Normal number B-cells but aka thymic aplasia velocardiofacial syndrome autosomal dominant CATCH-22 syndrome Cardiac defects tet fallot trunc art VSD Abnormal facial features micrognathia wide-spaced eyes low-set ears Thymic hypoplasia absent shadow T-cell deficiency Cleft palate craniofacial deformities Hypocalcemia hypoparathyroid seizures newborn tetany within chromosome 22q11 dev pharyngeal pouches Sx Infections viruses fungus Pneumocystis PCP PCP prophylaxis TMP/SMX bone marrow stem transplant Cure thymus SCID defect often adenosine deaminase failed development dysfunction Usually “bubble boy” no immune system starting 6 months Functionally AIDS Infections bacterial opportunistic PCP crypto candida toxo TB herpes chickenpox CMV PML diarrhea failure thrive Tx TMP/SMX English Wikipedia CC0 via Wikimedia Commons WAS protein Impaired cytoskeleton changes white cells platelets WIPE Infections thrombocytopenic Purpura Fig A Eczema B Bleeding hematoma A ear atopic diseases thrombocytopenia + splenectomy Low survival rates Cerebellar ataxia Incoordination stumbling falling slurred speech Oculocutaneous telangiectasia Recessive disease DNA repair predisp malignancy specific tx IVIG XLR NAPHD oxidase anion superoxides macrophages can ingest not kill catalase-positive organisms create granulomas instead respiratory GI UT skin infections granulomas ulcerations Catalase positive organisms Staph Aspergillus Burkholderia Nocardia Serratia Immune ↑ WBC can’t clear Measure neutrophil superoxide production Flow Dihydrorhodamine nitroblue tetrazolium test negative burst daily TMP/SMX antifungal itraconazole IFN-gamma CD18 integrin leukocyte chemotaxis Neutrophils adhere vessels exit impaired extravasation proliferates skin/mucosal skin GU pulm infections pus wounds high fever Newborns omphalitis inflammation umbilical stump delayed separation cord at 2-3 weeks old Lab WBC cytokines leukocytes high neutrophils Autosomal Lysosomes fuse together large granules neutrophils Peripheral neuropathy albinism skin eyes neutropenia for infections JAK-STAT signaling impairs Th17 proliferation FATED coarse Facies prominent forehead broad nose Abscesses cold noninflammatory containing S aureus Candida retained Teeth hyper-IgE Eosinophilia Derm severe eczema ↑↑ IgE eosinophilia Th17 otherwise membrane attack complex MAC which is required lysis Susceptible gram Neisseria meningococcal gonococcal antibiotics vaccines meningococcal Assembly MAC leads pores that disrupt target leading death hereditary angioedema AD deno excess bradykinin fluid accumulates mucosa trauma stress? ACEi NSAIDs Distinct from anaphylaxis no pruritus urticaria Not allergic mediated antihistamines don’t help GI abd pain n/v/d Resp laryngospasm obstruction increased risk but related
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This video “Primary Immunodeficiency: Introduction, Cellular Distribution & Background” is part of the Lecturio course “Immunology” ► WATCH the complete course on http://lectur.io/immunology6

► LEARN ABOUT:
– Introduction to Immunodeficiency
– Distribution of Cellular Primary Immunodeficiency Defects
– Primary Immunodeficiency Background
– X-Linked Primary Immunodeficiencies

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Peter Delves, Professor Emeritus of Immunology and former Vice Dean (Education) of the Faculty of Medical Sciences at University College London, is not only editor of two encyclopedias but also author of several textbooks and laboratory manuals. His special interest lies in improving an understanding of immunology through both web-based education and face-to-face interaction.

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“Examples of antibody deficiencies include when you have absent B cells such as X-linked agammaglobulinemia, impaired b cells such as common variable immune deficiency (CVID) or have selective antibody deficiency with abnormalities in shape and structure such as IgA deficiency.

For suspected antibody immunodeficiency disease important to measure immunoglobulins: Serum IgA, IgM and IgG subclasses and lymphocytes count and subsets. Remember to exclude secondary cause of low Immunoglobulins”

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Headline NTV/Politics – 21st January 2023:

Lauterbach warns of incurable immune deficiency caused by Corona
https://www.n-tv.de/politik/Lauterbach-warnt-vor-unheilbarer-Immunschwaeche-durch-Corona-article23860527.html
(Translate to English)

SARS-CoV-2 infection causes immunodeficiency in recovered patients by downregulating CD19 expression in B cells via enhancing B-cell metabolism
https://www.nature.com/articles/s41392-021-00749-3

The bigger question is, why now??

Read the explanation on Substack:
https://philipmcmillan.substack.com/p/warning-from-german-health-minister
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